Selected recent publications 

  1. Protein kinase C eta is activated in reactive astrocytes of an Alzheimer’s disease mouse model: Evidence for its immunoregulatory function in primary astrocytes. Muraleedharan A., et al., Glia. 2020 Oct 17.  
  2. Aging promotes reorganization of the CD4 T cell landscape toward extreme regulatory and effector phenotypes. Elyahu Y., et al., Sci Adv. 2019 Aug 21;5(8):eaaw8330. 
  3. CD4 T Cells Induce A Subset of MHCII-Expressing Microglia that Attenuates Alzheimer Pathology. Mittal K., et al., iScience. 2019 Jun 28;16:298-311.  
  4. BDNF-producing, amyloid β-specific CD4 T cells as targeted drug-delivery vehicles in Alzheimer’s disease. Eremenko E., et al., EBioMedicine. 2019 May;43:424-434.   
  5. The Choroid Plexus Functions as a Niche for T-Cell Stimulation Within the Central Nervous System.  Strominger I., et al., Front Immunol. 2018 May 16;9:1066. 
  6. Old Mice Accumulate Activated Effector CD4 T Cells Refractory to Regulatory T Cell-Induced Immunosuppression. Harpaz I., et al., Front Immunol. 2017 Mar 22;8:283. 
  7. TGF-β affinity-bound to a macroporous alginate scaffold generates local and peripheral immunotolerant responses and improves allocell transplantation. Orr S., et al., Acta Biomater. 2016 Nov;45:196-209. 
  8. Targeting innate immunity for neurodegenerative disorders of the central nervous system. Andreasson KI., J Neurochem. 2016 Sep;138(5):653-93. 
  9. Differential TGF-β Signaling in Glial Subsets Underlies IL-6-Mediated Epileptogenesis in Mice. Levy N., et al.,  J Immunol. 2015 Aug 15;195(4):1713-22.  
  10. Accelerated microglial pathology is associated with Aβ plaques in mouse models of Alzheimer’s disease. Baron R., et al., Aging Cell. 2014 Aug;13(4):584-95.